Rybakova, I.N., Humston, J.L., Sonnemann, K.J. and Ervasti, J.M. (2006) Dystrophin and utrophin bind actin filaments through distinct modes of contact. J. Biol. Chem. 281:9996-10001 (Published online 13 February 2006, 10.1074/jbc.M513121200). PDF

 

Hanft, L.M., Rybakova, I.N., Patel, J.R., Rafael, J.A. and Ervasti, J.M. (2006) Cytoplasmic γ-actin contributes to a compensatory remodeling response in dystrophin-deficient muscle. Proc. National Acad. Sci. USA 103:5385-5390 (Published online March 24, 2006 10.1073/pnas.0600980103). PDF

 

Sonnemann, K.J., Fitzsimons, D.P., Patel, J.R., Liu, Y.W., Schneider, M.F., Moss, R.L. and Ervasti, J.M. (2006) Cytoplasmic γ-actin is not required for skeletal muscle development but its absence leads to a progressive myopathy. Dev. Cell 11:387-397 (Published online September 5, 2006, DOI10.1016/j.devcel.2006.07.001). PDF

 

Ervasti, J.M. (2007) Dystrophin, its interactions with other proteins, and implications for muscular dystrophy. BBA Molec. Basis Dis. 1772:108-117 (Published online June 7, 2006 DOI 10.1016/j.bbadis.2006.05.010). PDF

 

Ervasti, J.M. and Sonnemann, K.J. (2008) Biology of the Dystrophin-Glycoprotein Complex. In: International Review of Cytology; A Survey of Cell Biology (K.W. Jeon, ed.) Ch. 5, pp. 191-225, Elsevier, San Diego. PDF

 

Prins K.W., Lowe D.A. and Ervasti, J.M. (2008) Skeletal muscle-specific ablation of γcyto-actin does not exacerbate the mdx phenotype. PLoS One 3:e2419. PDF

 

Bunnell, T.M., Jaeger M.A., Fitzsimons, D.P., Prins, K.W. and Ervasti, J.M. (2008) Destabilization of the dystrophin-glycoprotein complex without functional deficits in α-dystrobrevin null muscle PLoS One 3:e2604. PDF

 

Jaeger, M.A., Sonnemann, K.J., Fitzsimons, D., Prins, K.W. and Ervasti, J.M. (2009) Context dependent functional substitution of α-skeletal actin by γ-cytoplasmic actin FASEB J. 23:2205-2214. PDF

 

Prochniewicz, E., Henderson, D.M., Ervasti, J.M. and Thomas, D.D. (2009) Dystrophin and utrophin have distinct effects on the structural dynamics of actin Proc. National Acad. Sci. USA 106:7822-7827. PDF


Sonnemann, K.J., Heun-Johnson, H., Turner A.J., Baltgalvis, K., Lowe D.A. and Ervasti, J.M. (2009) Functional substitution by TAT-utrophin in dystrophin deficient mice. PLoS Med. 6:e1000083. PDF

 

Belyantseva, I.A., Perrin, B.J., Sonnemann, K.J., Zhu, M., Stepanyan, R., McGee, J., Frolenkov, G.A., Walsh, E.J., Friderici, K.H., Friedman, T.B. and Ervasti, J.M. (2009) γ-Actin is required for cytoskeletal maintenance but not development. Proc. National Acad. Sci. USA 106:9703-9708. PDF

 

Prins, K.W., Humston, J.M., Mehta, A., Tate, V., Ralston, E. and Ervasti, J.M. (2009) Dystrophin is a microtubule-associated protein J. Cell Biol. 186:363-369. PDF

 

Henderson, D.M., Lee, A. and Ervasti, J.M. (2010) Disease-Causing Missense Mutations in Actin Binding Domain 1 of Dystrophin Induce Thermodynamic Instability and Protein Aggregation. Proc. National Acad. Sci. USA 107:9632-9637. PDF

 

Bunnell, T.M. and Ervasti, J.M. (2010) Delayed embryonic development and impaired cell growth and survival in Actg1 null mice. Cytoskeleton 67:564-572. PDF

 

Perrin, B.J. and Ervasti, J.M. (2010) The actin gene family: function follows isoform. Cytoskeleton (In Press).

 

Perrin, B.J., Sonnemann, K.J and Ervasti, J.M. (2010) β-actin and γ-actin are each dispensible for auditory hair cell development but required for stereocilia maintenance. PLoS Genetics (In Press).